2 dysphagia 181014 revisi

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    181014 1

    CURRICULUM VITAE

    Full Name : MOHAMMAD SAIFUL-ISLAM doctor, neurologist, consultant (cerebroascular!

    "osition#$ob %itle : Medical Sta&&, Stro'e Unit, Deartment o&

    Neurolog), Facult) o& Medicine Airlangga Uniersit)#Dr Soetomo *eneral Hosital,

    Suraba)a

    +ducational ecords : Medical Doctor, Facult) o& Medicine AirlanggaUniersit), Suraba)a Secialist in Neurolog), Facult) o& Medicine, Airlangga Uniersit), Suraba)a %raining "rogram in te .linical Management o& Stro'e "atients at Sir .arles *airdner Hosital, "ert, /estern Australia (0112!

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    181014 FOEL 2

    NEUROLOGIC ASPECTON

    DYASPHAGIA

    Moammad Sai&ul-Islam

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    181014 FOEL 3

    Dysphagia is common and has !!n "!po"#!d #o !p"!s!n# in $%&' o( p!"sons ) ag! $*

    M!chanica+ dysphagia Oral: Am)loidosis, congenital

    abnormalities, Intraoral tumor, liin3uries, macroglosia,scleroderma, %M$ d)s&untion,4erostomia

    "ar)ngeal: .erical antosteo)tes, diteria,t)romegal), retroar)ngealabscess#tumor

    +soageal: .austic in3ur),esoageal .a#dierticulum#in&ection#stricture#ulceration,*+D, iatal ernia, osteriormediastinal mass

    Pfeiffer RS. NICP 2012;153-163

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    181014 FOEL 4

    Dysphagia is common and has !!n "!po"#!d #o !p"!s!n# in $%&' o( p!"sons ) ag! $*

    N!,"om,sc,+a"dysphagia

    Oroar)ngeal: in&lammator)m)osties, mitocondrialm)oat), muscular d)stro),NM$ disorders (M*, L+MS,botulism!, scleroderma, sti&& mans)ndrome

    +soageal: Am)loidosis,In&lammator) m)oaties(dermatom)ositis, ol)m)ositis,scleroderma

    Pfeiffer RS. NICP 2012;153-163

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    181014 FOEL 5

    Dysphagia is common and has !!n "!po"#!d #o !p"!s!n# in $%&' o( p!"sons ) ag! $*

    N!,"og!nic dysphagia

    Oroar)ngeal: Arnold-.iar)mal&ormation, basal gangliadiseases (DL5, HD, MSA, "D,

    "S", /ilson disease!, ."M, .",drug related, in&ection (rabies,encealitis, oliom)elitis!, masslesion (tumor, emorrage,abscess!, motor neuron disease(ALS!, MS, erieral neuroat)

    (*5S!, sinocerebellar ata4ia,s)ringobulbia, stro'e

    +soageal: Acalasia, autonomicneuroat), "D, esoagealmotilit) disorders, .agas diseasePfeiffer RS. NICP 2012;153-163

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    181014 FOEL 6

    IMPAIRED S-ALLO-ING OR DYSPHAGIAocc,"s .,i#! ("!.,!n#+y in n!,"o+ogica+ pa#i!n#s

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    181014 FOEL 7

    HO- COMMON ISDYSPHAGIA A/TER STRO0E 1

    Diagn!"i#"e#$ni%&e

    In#i'en#e f'(!)$agia

    C,"so"y

    sc"!!ning#!chni.,!

    37-45%

    C+inica+#!s#ing

    51-55%

    Ins#",m!n#a+#!s#ing 2V/S3

    64-78%

    *ar"in R e" a+. S"r,e 2005;362756-2763

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    181014 FOEL 8

    DOES DYSPHAGIA MATTER 1

    DYSPHAGIAIN ACUTE STRO0E PATIENTS IS A

    MAR0ER O/ POOR PROGNOSIS

    MALNUTRITIONDEHYDRATIONASPIRATIONPNEUMONIA

    PROLONGED HOSPITAL STAYINCREASED MOR4IDITY 5 MORTALITY

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    181014 FOEL

    DOES DYSPHAGIA MATTER 1

    S#,dy Dysphagic Non%Dysphagic

    RR 2"andom3

    6$' CI

    -!igh#2'3

    RR2"andom3

    6$' CI

    C$&a e" a+

    DePi)) e" a+

    /r'n e" a+

    /""i+e e" a+*ann e" a+

    Si"$ar' e" a+

    Sa+a e" a+

    To#a+5 CI

    621

    1082

    737

    502482

    2060

    1168

    &789**

    232

    157

    450

    130246

    57

    211

    :6896;

    7.4

    4.40

    13.2

    22.6.16

    34.05

    8.21

    ;**

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    181014 10

    O"opha"yng!a+ dysphagiais "!po"#!d ?i#h an incid!nc! o( 7*@;**' inpa#i!n#s ?i#h Pa">insons dis!as!

    C+ini#a++( re+ean" !a++ing

    i)airen" i! genera++(

    a!!#ia"e' i"$ a'an#e'

    !"age f "$e 'i!ea!e I" re'e! %&a+i"( f +ife

    #)+i#a"e! e'i#a"in in"a,e

    an' +ea'! " a+n&"ri"in an'

    a!)ira"in )ne&nia $i#$ i!a a9r #a&!e f 'ea"$ in

    Par,in!n:! 'i!ea!e

    Suntru S et al6 Brain 7809136; 726738

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    181014 FOEL 11

    NEURAL MECHANISMS O/ S-ALLO-ING

    Subcortical andcortical sensorimotor

    Medullar) s;allo;ing

    centerMotor outut (Nn6

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    181014 12

    NEURAL MECHANISMS O/ S-ALLO-ING

    BRAINSTEM

    S7a++7ingCen"e

    r

    THALAMUS

    SENSORI-MOTOR NEOCORTEX

    inferir )re- an' )!"-#en"ra+ g(r&!

    !+ihoodo( s?a++o?ing p"o+!ms

    Decrease LOC

    !""r s#$$#& 'a(ace D")'(e *e+#,ares#s O(er a&e A'"r+a( $"&)e,a(a$a( +".e+e$ /ea0 "r a'se$ ."()$ar c")&*

    M"#s$ "r ')''(#& ."#ce E.#ece "2 ,e)+"#a Re)ce ,*ar&ea( sesa$#"

    ESOPHAGEALPHASE

    P"ima"y #!s#

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    ASSESSMENT O/ S-ALLO-ING

    B CLINICAL /EATURES

    D)soniaD)sartriaAbnormal gag re&le4Abnormal olitional coug

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    181014 FOEL 1

    MANAGEMENT

    GOALS

    "reention o&asiration,

    de)dration,malnutrition

    eabilitation o&d)s&unction

    eestablisment o&oral inta'e

    COMPENSATORYSTRATEGY

    %o 'ee atients sa&e

    ;en eating

    REHA4ILITATIVE

    STRATEGY %o seed te recoer)rocess

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    COMPENSATORY TREATMENT

    Nil b) mout

    (N5M! N* and "+*

    &eeding

    Feeding

    training

    Ne&r+gi#a+ ar' Dr Se" !)i"a+ S&raa(a D#.

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    SUMMARY

    Neurogenicd)sagia

    .ommon roblemin neurologicaldiseases

    Neural

    mecanismsAssessmentManagement

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    180512 FOEL 22220711 FOEL 22

    22

    Suramadu Long Bridge(Surabaya-Madura)

    THAN0 YOU